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Physio: 7# Neurophysiology of Vision

Q21. The immediate effect of light on photoreceptors is:

Depolarization
Hyperpolarization
Action potential generation
Neurotransmitter release
Explanation:
Light closes Na⁺ channels causing hyperpolarization.

Q22. In darkness, photoreceptors are:

Hyperpolarized
Silent
Depolarized and releasing inhibitory neurotransmitter
Releasing excitatory neurotransmitter
Explanation:
Dark current keeps photoreceptors depolarized.

Q23. Closure of sodium channels during phototransduction is due to:

↓ cGMP
↑ cGMP
↑ calcium
↓ phosphodiesterase
Explanation:
Light activates PDE → ↓ cGMP → Na⁺ channel closure.

Q24. Vitamin A deficiency leads to:

Color blindness
Glaucoma
Macular degeneration
Night blindness
Explanation:
Vitamin A is essential for rhodopsin regeneration.

Q25. Complete dark adaptation of rods takes approximately:

5–10 minutes
30–50 minutes
1–2 minutes
Several hours
Explanation:
Rods adapt slower but reach higher sensitivity.

Q26. Y ganglion cells are specialized for:

Rapid movement detection
Color discrimination
Fine visual acuity
Night vision only
Explanation:
Y cells transmit fast, black-and-white motion information.

Q27. Fibers from the nasal retina:

Remain ipsilateral
Terminate in superior colliculus only
Decussate at optic chiasm
Terminate in cerebellum
Explanation:
Nasal retinal fibers cross at the optic chiasm.

Q28. Lesion at the optic chiasm causes:

Homonymous hemianopia
Quadrantanopia
Central scotoma
Bitemporal hemianopia
Explanation:
Crossing nasal fibers carry temporal visual fields.

Q29. The primary relay station for vision is the:

Superior colliculus
Lateral geniculate body
Pretectal nucleus
Visual cortex
Explanation:
LGB controls and relays signals to visual cortex.

Q30. Magnocellular layers of LGB receive input from:

Y ganglion cells
X ganglion cells
W ganglion cells
Amacrine cells
Explanation:
Magnocellular layers (1–2) process motion & black-white vision.

Q31. Parvocellular layers of LGB are responsible for:

Motion detection
Night vision
Color and fine detail
Reflex eye movement
Explanation:
Layers 3–6 process color and high acuity.

Q32. Primary visual cortex corresponds to:

Area 18
Area 17
Area 19
Area 6
Explanation:
Area 17 lies in the calcarine fissure.

Q33. The fourth layer of visual cortex primarily receives input from:

Association cortex
Superior colliculus
Optic nerve
Lateral geniculate body
Explanation:
Layer IV is the main thalamocortical input layer.

Q34. The dorsal visual stream answers:

Where is the object?
What is the object?
What color is it?
Is it familiar?
Explanation:
Dorsal stream processes motion and spatial location.

Q35. The ventral visual stream is mainly concerned with:

Motion
Depth
Object recognition
Eye movement
Explanation:
Ventral stream is the “what” pathway.

Q36. Damage to inferior temporal cortex causes inability to:

See motion
See color
See light
Recognize faces
Explanation:
IT cortex is critical for face recognition.

Q37. The suprachiasmatic nucleus receives fibers mainly for:

Visual perception
Circadian rhythm control
Pupillary reflex
Eye movements
Explanation:
SCN is the biological clock.

Q38. Pretectal nucleus is essential for:

Pupillary light reflex
Visual acuity
Color vision
Depth perception
Explanation:
Pretectal nucleus mediates pupillary reflexes.

Q39. Superior colliculus is involved in:

Color discrimination
Form vision
Rapid eye movements
Visual memory
Explanation:
Superior colliculus controls rapid, reflex eye movements.

Q40. Contralateral homonymous hemianopia results from lesion of:

Optic nerve
Optic chiasm
Retina
Optic radiation
Explanation:
Optic radiation lesions affect same visual field in both eyes.